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Hey, how is it going?
I found an issue when comparing the following 2 HTML texts:
First:
`
The study of diabetes and its complications in experimental models, is overseen by the Animal Models of Diabetic Complications Consortium (AMDCC). They have approved and recommended the induction of diabetes in mice using the low-dose injection of streptozotocin (STZ, 55mg/kg/day) over five days. STZ causes the destruction of pancreatic beta cells, ultimately resulting the the lack of insulin production thus establishing a model of insulin deficient diabetes. Diabetic mice will develop diabetes within 1 week of completing STZ injections. Phenotypically, the diabetic mice will have a slower rate of weight gain, increased appetite, increased thirst and increased production of urine.
Apoe -/- mice are the genetic model for the study of atherosclerosis. Diabetes will be induced in these mice with slow dose STZ (5 daily injections of 55/mg KG BW) and within 10 weeks mice significanlty develop atherosclerosis.
<table>
<tbody>
<tr>
<td>Strain</td>
<td>Description</td>
<td>Phenotype</td>
<td>Health status</td>
<td>Purpose of use</td>
</tr>
<tr>
<td>1. ApoE -/-</td>
<td>Apoe -/- mice are the genetic model to study atherosclerosis. They develop atherosclerosis upon STZ induced diabetes</td>
<td>Traditional black coated mouse as per the C57Bl/6 mouse</td>
<td>This strain is the same as the traditional C57Bl/6 mouse.</td>
<td>To test if AP-1 inhibition by T-5224 can attenuate atherosclerosis.</td>
</tr>
</tbody>
</table>`
Second:
`
The study of diabetes and its complications in experimental models, is overseen by the Animal Models of Diabetic Complications Consortium (AMDCC). They have approved and recommended the induction of diabetes in mice using the low-dose injection of streptozotocin (STZ, 55mg/kg/day) over five days. STZ causes the destruction of pancreatic beta cells, ultimately resulting the the lack of insulin production thus establishing a model of insulin deficient diabetes.
<table>
<tbody>
<tr>
<td>Strain</td>
<td>Description</td>
<td>Phenotype</td>
<td>Health status</td>
<td>Purpose of use</td>
</tr>
<tr>
<td>1.ApoE -/-</td>
<td>ApoE -/- mice are the genetic model to study atherosclerosis. Diabetes will be induced in these mice with slow dose STZ (5 daily injections of 55/mg KG BW mae and 75 mg/Kg BW female) and within 10 weeks mice significantly develop atherosclerosis. Five weeks post diabetes, mice will receive T-5224 for 5 weeks.</td>
<td><br>We do not expect any visible phenotypic changes as the experiment progresses.</td>
<td>Diabetic mice will develop diabetes within 2 weeks of completing STZ injections. Health wise, the diabetic mice will have a slower rate of weight gain, increased appetite, increased thirst and increased production of urine.We do not expect any changes in health status with T-5224 treatment.</td>
<td>To test if AP-1 inhibition by T-5224 can attenuate atherosclerosis in diabetes.</td>
</tr>
</tbody>
</table>`
This is the resulting HTML:
`
The study of diabetes and its complications in experimental models, is overseen by the Animal Models of Diabetic Complications Consortium (AMDCC). They have approved and recommended the induction of diabetes in mice using the low-dose injection of streptozotocin (STZ, 55mg/kg/day) over five days. STZ causes the destruction of pancreatic beta cells, ultimately resulting the the lack of insulin production thus establishing a model of insulin deficient diabetes.
| Strain | Description | Phenotype | Health status | Purpose of use | |||||||||
| 1.ApoE -/- | ApoE -/- mice are the genetic model to study atherosclerosis. Diabetes will be induced in these mice with slow dose STZ (5 daily injections of 55/mg KG BW mae and 75 mg/Kg BW female) and within 10 weeks mice significantly develop atherosclerosis. Five weeks post diabetes, mice will receive T-5224 for 5 weeks. | We do not expect any visible phenotypic changes as the experiment progresses. | Diabetic mice will develop diabetes within not
It is somehow generating 2 tables and one of them has missing |
This is how it looks like in the UI:

Thanks for taking the time to have a look at this!
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